Myocytolysis occurs during muscle fatigue, leading to the breakdown of muscle fibers.
在肌肉疲劳过程中会发生肌细胞溶解,导致肌肉纤维的分解。
The necrotic
myocytolysis seen in muscular dystrophies is a hallmark of the disease.
肌肉萎缩症中的坏死性肌细胞溶解是该疾病的一个特征标志。
Exercise-induced
myocytolysis contributes to the adaptive response of muscle growth.
运动引起的肌细胞溶解对肌肉生长的适应性反应有所贡献。
Myocytolysis releases creatine kinase into the bloodstream, which can be detected in a blood test.
肌细胞溶解会释放肌酸激酶进入血液,这可以在血液检测中被发现。
Inflammatory cytokines can trigger
myocytolysis, exacerbating muscle damage after injury.
炎症细胞因子能引发肌细胞溶解,加剧损伤后的肌肉损害。
Muscle biopsies revealed extensive
myocytolysis and inflammation among patients with polymyositis.
对于多发性肌炎患者,肌肉活检显示出广泛的肌细胞溶解和炎症。
Myocytolysis is a protective mechanism that clears damaged muscle fibers, allowing regeneration to occur.
肌细胞溶解是一种保护机制,清除受损的肌肉纤维,以便再生。
The recovery process after intense exercise includes repair of
myocytolysis-induced damage.
强度运动后的恢复过程包括修复由肌细胞溶解引起的损伤。
Researchers studying muscle fatigue observed increased
myocytolysis in response to prolonged exercise.
研究肌肉疲劳的研究人员观察到在长时间锻炼后肌细胞溶解的增加。
Early detection of
myocytolysis through biomarkers can aid in diagnosing and managing muscular disorders.
通过生物标志物早期检测肌细胞溶解有助于诊断和管理肌肉障碍。
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